| Archives of Razi Institute | ||
| Article 21, Volume 78, Issue 5, January and February 0, Pages 1594-1602 PDF (354.4 K) | ||
| Document Type: مقالات پژوهشی | ||
| DOI: 10.32592/ARI.2023.78.5.1594 | ||
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Serotonin produces monoamine oxidase-dependent oxidative stress in human heart valves. American Journal of Physiology-Heart and Circulatory Physiology. 2009;297(4):H1354-H60. 6. Gustafsson BrI, Tømmerås K, Nordrum I, Loennechen JP, Brunsvik A, Solligård E, et al. Long-term serotonin administration induces heart valve disease in rats. Circulation. 2005;111(12):1517-22. 7. Elangbam CS, Job LE, Zadrozny LM, Barton JC, Yoon LW, Gates LD, et al. 5-hydroxytryptamine (5HT)- induced valvulopathy: compositional valvular alterations are associated with 5HT2B receptor and 5HT transporter transcript changes in Sprague-Dawley rats. Experimental and Toxicologic Pathology. 2008;60(4-5):253-62. 8. Nebigil CG, Désaubry L. The role of GPCR signaling in cardiac Epithelial to Mesenchymal Transformation (EMT). Trends in Cardiovascular Medicine. 2019;29(4):200-4. 9. Mialet-Perez J, Santin Y, Parini A. 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Obesity-associated cardiac pathogenesis in broiler breeder hens: Pathological adaption of cardiac hypertrophy1,2. Poultry Science. 2017;96(7):2428-37. 14. Zhang J, Schmidt CJ, Lamont SJ. Transcriptome analysis reveals potential mechanisms underlying differential heart development in fast-and slow-growing broilers under heat stress. BMC genomics. 2017;18(1):1- 15. 15. Brasil D, Temsah RM, Kumar K, Kumamoto H, Takeda N, Dhalla NS. Blockade of 5-HT2A receptors by sarpogrelate protects the heart against myocardial infarction in rats. J Cardiovasc Pharmacol Ther. 2002;7(1):53-9. 16. Miyata K, Shimokawa H, Higo T, Yamawaki T, Katsumata N, Kandabashi T, et al. Sarpogrelate, a selective 5-HT2A serotonergic receptor antagonist, inhibits serotonin-induced coronary artery spasm in a porcine model. J Cardiovasc Pharmacol. 2000;35(2):294- 301. 17. Neri-Gómez T, Valero-Elizondo G, MansillaOlivares A, Mondragón-Herrera JA, Manjarrez-Gutiérrez G. 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Nature Communications. 2020;11(1):4364. 22. Mendes P, Martinho R, Leite S, Maia-Moço L, Leite-Moreira AF, Lourenço AP, et al. Chronic exercise induces pathological left ventricular hypertrophy in adrenaline-deficient mice. Int J Cardiol. 2018;253:113-9. 23. Wong DL, Tai T, Wong-Faull DC, Claycomb R, Meloni EG, Myers KM, et al. Epinephrine: A short-and long-term regulator of stress and development of illness. Cell Mol Neurobiol. 2012;32(5):737-48. 24. Zhang DY, Anderson AS. The sympathetic nervous system and heart failure. Cardiol Clin. 2014;32(1):33-45. 25. Cases O, Seif I, Grimsby J, Gaspar P, Chen K, Pournin S, et al. Aggressive behavior and altered amounts of brain serotonin and norepinephrine in mice lacking MAOA. Science. 1995;268(5218):1763-6. 26. Sivasubramaniam S, Finch C, Rodriguez M, Mahy N, Billett E. A comparative study of the expression of monoamine oxidase-A and-B mRNA and protein in nonCNS human tissues. Cell Tissue Res. 2003;313(3):291- 300. 27. Lairez O, Calise D, Bianchi P, Ordener C, SpreuxVaroquaux O, Guilbeau-Frugier C, et al. Genetic deletion of MAO-A promotes serotonin-dependent ventricular hypertrophy by pressure overload. J Mol Cell Cardiol. 2009;46(4):587-95. 28. Jeong S-J, Park J-G, Oh GT. Peroxiredoxins as Potential Targets for Cardiovascular Disease. Antioxidants [Internet]. 2021; 10(8). | ||
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