اخبار و اعلانات

 آمار

تعداد نشریات282
تعداد نشریات سازمان80
تعداد شماره‌ها3,208
تعداد مقالات35,560
تعداد مشاهده مقاله53,003,530
تعداد دریافت فایل اصل مقاله92,288,578
Tretyakova, V. A, Zhernakova, N. I, Arisheva, O. S, Garmash, I. V, Tretyakov, A, Gerasimov, N. I, Ermilov, O. V. (1400). Meta-Analysis of Non-Alcoholic Fatty Liver Disease and Electromechanical Reconstruction of Myocardium. سامانه مدیریت نشریات علمی, 77(1), 135-140. doi: 10.22092/ari.2021.356231.1808
V. A Tretyakova; N. I Zhernakova; O. S Arisheva; I. V Garmash; A Tretyakov; N. I Gerasimov; O. V Ermilov. "Meta-Analysis of Non-Alcoholic Fatty Liver Disease and Electromechanical Reconstruction of Myocardium". سامانه مدیریت نشریات علمی, 77, 1, 1400, 135-140. doi: 10.22092/ari.2021.356231.1808
Tretyakova, V. A, Zhernakova, N. I, Arisheva, O. S, Garmash, I. V, Tretyakov, A, Gerasimov, N. I, Ermilov, O. V. (1400). 'Meta-Analysis of Non-Alcoholic Fatty Liver Disease and Electromechanical Reconstruction of Myocardium', سامانه مدیریت نشریات علمی, 77(1), pp. 135-140. doi: 10.22092/ari.2021.356231.1808
Tretyakova, V. A, Zhernakova, N. I, Arisheva, O. S, Garmash, I. V, Tretyakov, A, Gerasimov, N. I, Ermilov, O. V. Meta-Analysis of Non-Alcoholic Fatty Liver Disease and Electromechanical Reconstruction of Myocardium. سامانه مدیریت نشریات علمی, 1400; 77(1): 135-140. doi: 10.22092/ari.2021.356231.1808

Meta-Analysis of Non-Alcoholic Fatty Liver Disease and Electromechanical Reconstruction of Myocardium

Archives of Razi Institute
مقاله 20، دوره 77، شماره 1، فروردین و اردیبهشت 2022، صفحه 135-140    اصل مقاله (406.9 K)
نوع مقاله: Original Articles
شناسه دیجیتال (DOI): 10.22092/ari.2021.356231.1808
نویسندگان
V. A Tretyakova* 1، 2؛ N. I Zhernakova1؛ O. S Arisheva2؛ I. V Garmash2؛ A Tretyakov1؛ N. I Gerasimov1؛ O. V Ermilov1
1Department of Medicine and Pediatrics, Medical Institute, Belgorod State University, Belgorod, Russia
2Department of Internal Medicine with a Course of Cardiology and Functional Diagnostics Named after V. S. Moiseev., Institution of Higher Professional Education, "Peoples' Friendship University of Russia" Moscow, Russia
چکیده
In developed countries, non-alcoholic fatty liver disease (NAFLD), which results from obesity, has become endemic and kills many adults annually. Health research centers in most countries are looking to examine the relationship between metabolic syndrome (MetS) and metabolic biomarkers. A bleeding-prone standard liver biopsy or costly magnetic resonance imaging scan is used to diagnose NAFLD. The present study aimed to analyze medical databases using various scientific articles; moreover, this experiment analyzed medical databases using published scientific articles related to NAFLD, endovascular treatment, cardiac arrhythmias and conduction disorders, changes in the geometry of atria and ventricles, changes in myocardial mass volume as well as diastolic flow left and right ventricular systolic functions, coronary blood flow, analysis of the dependence of epicardial fat tissue (EFT) thickness, and the presence of chronic heart failure (CHF). It is demonstrated that the index of EFT in NAFLD positively correlated with the criteria of cardiovascular health, values of the carotid intima-media thickness, and calcification of the coronary arteries on the coronary artery calcium scale (P<0.0001). The index of per EFT significantly correlated with the factor of the age of the NAFLD patients (P=0.04), hemoglobin A1C level (P<0.001), systemic inflammatory index (P=0.02), the index of impaired glucose tolerance (P=0.03), and especially, the patient's diabetes factor (P<0.001). In addition, adiponectin levels were significantly lower in individuals with NAFLD (P=0.001) and patients with MetS (P=0.02). NAFLD in association with an increase in epicardial adipose tissue (EAT) is an independent risk factor for atherosclerosis, coronary heart disease, CHF, as well as structural and electrophysiological myocardial remodeling. The study of pathogenetic mechanisms in the context of the role of EAT and clinical monitoring of its condition are urgent problems of modern medicine.
کلیدواژه‌ها
Epicardial adipose tissue؛ Electrophysiological؛ Myocardial remodeling؛ Non-alcoholic fatty liver
سایر فایل های مرتبط با مقاله
مراجع
  1. Neuman MG. Cytokines—central factors in alcoholic liver disease. Alcohol Res Health. 2003;27(4):307.
  2. Kawano Y, Cohen DE. Mechanisms of hepatic triglyceride accumulation in non-alcoholic fatty liver disease. J Gastroenterol. 2013;48(4):434-41.
  3. Chen M, Guo W-L, Li Q-Y, Xu J-X, Cao Y-J, Liu B, et al. The protective mechanism of Lactobacillus plantarum FZU3013 against non-alcoholic fatty liver associated with hyperlipidemia in mice fed a high-fat diet. Food Funct. 2020;11(4):3316-31.
  4. Fan JG, Cao HX. Role of diet and nutritional management in non‐alcoholic fatty liver disease. J Gastroenterol Hepatol. 2013;28:81-7.
  5. Neuschwander-Tetri BA, Caldwell SH. Nonalcoholic steatohepatitis: summary of an AASLD Single Topic Conference. J Hepatol. 2003;37(5):1202-19.
  1. Sohrabpour AA, Rezvan H, Amini-Kafiabad S, Dayhim M, Merat S, Pourshams A. Prevalence of nonalcoholic steatohepatitis in Iran: a population based study. Middle East J Dig Dis. 2010;2(1):14.
  2. Lankarani KB, Ghaffarpasand F, Mahmoodi M, Lotfi M, Zamiri N, Heydari ST, et al. Non alcoholic fatty liver disease in southern Iran: a population based study. Hepat Mon. 2013;13(5).
  3. Martín-Domínguez V, González-Casas R, Mendoza-Jiménez-Ridruejo J, García-Buey L, Moreno-Otero R. Pathogenesis, diagnosis and treatment of non-alcoholic fatty liver disease. Rev Esp Enferm Dig. 2013;105(7):409-20.
  4. Jamali R, Khonsari M, Merat S, Khoshnia M, Jafari E, Kalhori AB, et al. Persistent alanine aminotransferase elevation among the general Iranian population: prevalence and causes. World J Gastroenterol. 2008;14(18):2867.
  5. Le MH, Devaki P, Ha NB, Jun DW, Te HS, Cheung RC, et al. Prevalence of non-alcoholic fatty liver disease and risk factors for advanced fibrosis and mortality in the United States. PloS One. 2017;12(3): 0173499.
  6. Cotter TG, Rinella M. Nonalcoholic fatty liver disease 2020: the state of the disease. Gastroenterology. 2020;158(7):1851-64.
  7. Psychari SN, Rekleiti N, Papaioannou N, Varhalama E, Drakoulis C, Apostolou TS, et al. Epicardial fat in nonalcoholic fatty liver disease: properties and relationships with metabolic factors, cardiac structure, and cardiac function. Angiology. 2016;67(1):41-8.
  8. Moore JX, Chaudhary N, Akinyemiju T. Metabolic Syndrome Prevalence by Race/Ethnicity and Sex in the United States, National Health and Nutrition Examination Survey, 1988-2012. Prev Chronic Dis. 2017;14:24..
  9. Mottillo S, Filion KB, Genest J, Joseph L, Pilote L, Poirier P, et al. The metabolic syndrome and cardiovascular risk: a systematic review and meta-analysis. J Am Coll Cardiol. 2010;56(14):1113-32.
  10. Chang C-C, Huang Z-Y, Shih S-F, Luo Y, Ko A, Cui Q, et al. Electrical impedance tomography for non-invasive identification of fatty liver infiltrate in overweight individuals. Sci Rep. 2021;11(1):19859.
  11. Sacks HS, Fain JN. Human epicardial adipose tissue: a review. Am Heart J. 2007;153(6):907-17.
  12. Iacobellis G, Pistilli D, Gucciardo M, Leonetti F, Miraldi F, Brancaccio G, et al. Adiponectin expression in human epicardial adipose tissue in vivo is lower in patients with coronary artery disease. Cytokine. 2005;29(6):251-5.
  1. Xu L, Yan J, Zhang F, Zhou C, Fan T, Chen X, et al. Use of inflammatory biomarkers and real-time cardiac catheterisation to evaluate the left ventricular diastolic function in patients with diastolic heart failure. Heart Lung Circ. 2021;30(3):396-403.
  2. Iacobellis G, Leonetti F, Singh N, Sharma AM. Relationship of epicardial adipose tissue with atrial dimensions and diastolic function in morbidly obese subjects. Int J Cardiol. 2007;115(2):272-3.
  3. Blumensatt M, Fahlbusch P, Hilgers R, Bekaert M, De Wiza DH, Akhyari P, et al. Secretory products from epicardial adipose tissue from patients with type 2 diabetes impair mitochondrial β-oxidation in cardiomyocytes via activation of the cardiac renin–angiotensin system and induction of miR-208a. Basic Res. Cardiol. 2017;112(1):1-13.
  4. Chechi K, Voisine P, Mathieu P, Laplante M, Bonnet S, Picard F, et al. Functional characterization of the Ucp1-associated oxidative phenotype of human epicardial adipose tissue. Sci Rep. 2017;7(1):1-15.
  5. Farias-Itao DS, Pasqualucci CA, Nishizawa A, Silva LFF, Campos FM, Da Silva KCS, et al. Perivascular adipose tissue inflammation and coronary artery disease: an autopsy study protocol. JMIR Res Protoc. 2016;5(4):e211.
  6. Noyes AM, Dua K, Devadoss R, Chhabra L. Cardiac adipose tissue and its relationship to diabetes mellitus and cardiovascular disease. World J Diabetes. 2014;5(6):868.
  7. Arshad T, Golabi P, Henry L, Younossi ZM. Epidemiology of non-alcoholic fatty liver disease in North America. Curr Pharm Des. 2020;26(10):993-7.
  8. Younossi ZM, Koenig AB, Abdelatif D, Fazel Y, Henry L, Wymer M. Global epidemiology of nonalcoholic fatty liver disease—meta‐analytic assessment of prevalence, incidence, and outcomes. Hepatology. 2016;64(1):73-84.
  1. Younossi ZM, Marchesini G, Pinto-Cortez H, Petta S. Epidemiology of nonalcoholic fatty liver disease and nonalcoholic steatohepatitis: implications for liver transplantation. Transplantation. 2019;103(1):22-7.
  2. Christensen RH, von Scholten BJ, Lehrskov LL, Rossing P, Jørgensen PG. Epicardial adipose tissue: an emerging biomarker of cardiovascular complications in type 2 diabetes? Ther Adv Endocrinol Metab. 2020;11:2042018820928824.
  3. Mitra S, De A, Chowdhury A. Epidemiology of non-alcoholic and alcoholic fatty liver diseases. Transl Gastroenterol Hepatol. 2020;5.
  4. Meng X, Wang W, Zhang K, Qi Y, An S, Wang S, et al. Epicardial adipose tissue volume is associated with non-alcoholic fatty liver disease and cardiovascular risk factors in the general population. Ther Clin Risk Manag. 2018;14:1499.
  5. Wu M-Z, Lee C-H, Chen Y, Yu S-Y, Yu Y-J, Ren Q-W, et al. Association between adipocyte fatty acid-binding protein with left ventricular remodelling and diastolic function in type 2 diabetes: a prospective echocardiography study. Cardiovasc Diabetol. 2020;19(1):1-11.
  6. McKenney ML, Schultz KA, Boyd JH, Byrd JP, Alloosh M, Teague SD, et al. Epicardial adipose excision slows the progression of porcine coronary atherosclerosis. J Cardiothorac Surg. 2014;9(1):1-11.
  7. Santiago-Fernández C, Pérez-Belmonte LM, Millán-Gómez M, Moreno-Santos I, Carrasco-Chinchilla F, Ruiz-Salas A, et al. Overexpression of scavenger receptor and infiltration of macrophage in epicardial adipose tissue of patients with ischemic heart disease and diabetes. J Transl Med. 2019;17(1):1-10.
آمار
تعداد مشاهده مقاله: 3,613
تعداد دریافت فایل اصل مقاله: 4,473


counter
سامانه مدیریت نشریات علمی. طراحی و پیاده سازی از سیناوب